Examples of 'cardiac toxicity' in a sentence
Meaning of "cardiac toxicity"
Cardiac toxicity refers to the harmful or damaging effects that certain substances or medications can have on the heart. These substances may interfere with the normal functioning of the heart muscle, electrical conduction system, or blood vessels, leading to adverse cardiovascular events or conditions. Cardiac toxicity can occur as a side effect of certain drugs or as a result of exposure to toxins or chemicals
How to use "cardiac toxicity" in a sentence
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cardiac toxicity
There are many possible causes of cardiac toxicity.
Cardiac toxicity is also an important consideration in evaluating drug candidates.
There was no clinically significant cardiac toxicity.
Oxaliplatin cardiac toxicity was the most frequent cause of lethal events observed in dogs.
The condition may be cardiac toxicity.
The cardiac toxicity may be associated with the administration of a drug to the individual.
It has not been associated with cardiac toxicity or electrocardiographic changes.
These grading scores are consistent with no or mild cardiac toxicity.
The cardiac toxicity induced by intracoronary injection in sheep was similar for the three drugs.
There was no exacerbation of cardiac toxicity.
Methods for predicting cardiac toxicity before and upon treatment with tyrosine kinase inhibitors.
Damage to the heart muscle by a toxin is called cardiac toxicity.
Cardiac toxicity Doxorubicin and other anthracyclines can cause cardiotoxicity.
Special attention must be given to the cardiac toxicity exhibited by doxorubicin.
More patients are living long enough to experience this risk of cardiac toxicity.
See also
Cardiac toxicity with Pixuvri may occur whether or not cardiac risk factors are present.
Interaction with the hERG channel is one indicator of potential cardiac toxicity.
In adult patients severe cardiac toxicity may occur precipitously without antecedent ECG changes.
Measuring cardiac cell contraction to identify new therapies and predict cardiac toxicity.
In addition, cardiac toxicity is a unique adverse characteristic of anthracycline antibiotics.
We observed a reduction in treatment time and less cardiac toxicity in the pentamidine group.
Cardiac toxicity may be caused by medications, usually ones used to treat cancer.
Cardiac function should be carefully monitored during treatment to minimize the risk of cardiac toxicity.
Thioridazine HCl can be toxic in overdose, with cardiac toxicity being of particular concern.
The risk of cardiac toxicity rises with increasing cumulative doses of anthracyclines, including doxorubicin.
The use of DOXIL may lead to cardiac toxicity.
After that, cardiac toxicity is treated the same as heart failure from other causes.
No, can induce from a condition to cardiac toxicity.
Summary of Cardiac Toxicity Based on preclinical studies, oxaliplatin is cardiotoxic.
In some embodiments, the condition is cardiac toxicity.
Possible cardiac toxicity ( tachycardia and decreased contractility ) . Occasionally, a transient reduction in.
This medication is effective, but is limited by cardiac toxicity.
Any grade 3 or 4 drug-related non cardiac toxicity other than nausea or vomiting.
There have been many fatalities, particularly due to cardiac toxicity.
Some conditioning regimens may have inherent cardiac toxicity with long-term cardiac consequences.
Trastuzumab-based therapy is also associated with potential cardiac toxicity.
Researchers identified patients who developed cardiac toxicity -- asymptomatic, symptomatic, or both.
Pegylated liposomal doxorubicin has shown a similar anti-cancer effect to doxorubicin, but with less cardiac toxicity.
For leishmaniasis, the drugs cause renal and cardiac toxicity and trigger resistance.
Combining mitoxantrone with potentially cardiotoxic active substances ( e.g. anthracyclines ) increases the risk of cardiac toxicity.
Cardiac toxicity accounts for late deaths in 2 % autologous and 3 % of allogeneic HCT recipients.
In addition, their activity is more stable and their cardiac toxicity is lower.
In particular, no hepatic toxicity, cardiac toxicity ( ECG ) or renal toxicity ( creatininemia ) was observed.
As a result, ECG levels are unknown in the prediction of cardiac toxicity.
Mg/m2/every 3 weeks met the protocol-defined criteria for cardiac toxicity during treatment and / or follow-up.
However, reduction of the QRS complex is considered more indicative of cardiac toxicity.
An independent retrospective study described also significant cardiac toxicity in 4 out of 8 patients.
For dose reduction of Myocet due to liver function impairment, see section 4.2 . Cardiac toxicity.
Consequently, the value pill of the ECG in predicting cardiac toxicity is unknown.
High selectivity for the H1 receptor and no cardiac toxicity.
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Examples of using Cardiac
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Cardiac neurological effects have also been reported
Disturbances of cardiac rhythm and arrhythmia devices
Cardiac arrest for at least two hours
Examples of using Toxicity
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Adequate chronic toxicity data not available
Toxicity is a measure of how harmful or poisonous a pesticide is
No clinical signs of toxicity were observed at any dose