Examples of 'excitotoxicity' in a sentence
Meaning of "excitotoxicity"
excitotoxicity (noun): damage or destruction of nerve cells due to excessive stimulation by neurotransmitters
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- The continuous stimulation of a nerve cell by a neurotransmitter, especially by glutamates
How to use "excitotoxicity" in a sentence
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excitotoxicity
We have spoken about the excitotoxicity of glutamate.
Purkinje cells are especially susceptible to ethanol excitotoxicity.
The phenomenon of excitotoxicity is no longer observed at this stage.
Neuronal death due to overstimulation is called excitotoxicity.
Glutamate induced excitotoxicity causes an elevation of intracellular calcium.
Changes in calcium flux and excitotoxicity.
Damaging excitotoxicity may also occur as a result of repeated withdrawals.
Hypoglycemia is the main cause of excitotoxicity.
This is a condition called excitotoxicity of the glutamate receptors.
Overload of calcium is a key process of excitotoxicity.
Excitotoxicity is phenomenon in which glutamate receptors are inappropriately activated.
Researchers also speculate that it may inhibit glutamate excitotoxicity.
The process of excitotoxicity has been extensively studied in the retina.
This phenomenon is known as excitotoxicity.
This is excitotoxicity.
See also
There is described treating or effecting prophylaxis of a disease mediated by excitotoxicity.
White matter excitotoxicity.
Several glutamate receptors antagonists have been tested to counteract excitotoxicity.
This suggested that glutamate excitotoxicity was implicated in ethambutol mediated cell loss.
Studies have shown that antioxidants are powerful protectors against excitotoxicity.
Another approach in limiting excitotoxicity consists in inhibiting the presynaptic release of glutamate.
One of the most documented cases relates to the excitotoxicity of glutamate.
Excitotoxicity can occur from substances produced within the body endogenous excitotoxins.
The process of neural degeneration is often the result of glutamate excitotoxicity.
Neuronal cell depolarization can lead to excitotoxicity and neuronal cell death.
This combination of neuroexcitatory activities and neurotoxic properties is called excitotoxicity.
Its neuroprotective properties in the case of glutamate excitotoxicity are demonstrated in cell culture.
One of the proposed mechanisms downstream from a hi event is called glutamatergic excitotoxicity.
These results suggest that excitotoxicity might not be the mecanism of motoneuron death.
The cause of neuron cell death in a number of neurodegenerative diseases is due to excitotoxicity.
Excitatory amino acid excitotoxicity has been implicated in the pathophyslology of a number of neurological disorders.
There is also evidence that several nutrients can significantly reduce excitotoxicity.
Extensive evidence supports that excitotoxicity also contributes to neuronal death in neurodegenerative diseases.
Glutamine synthetase downregulation reduces astrocyte protection against glutamate excitotoxicity to neurons.
Excitotoxicity consists in an uncontrolled synaptic overactivity leading to the enhanced release of excitatory neurotransmitters.
Activating these receptors contributes to synaptic plasticity in some circumstances and excitotoxicity in others.
Excitatory amino acid excitotoxicity has been implicated in the pathophysiology of numerous neurological disorders.
Tau reduction also protected both transgenic and nontransgenic mice against excitotoxicity.
This excitotoxicity has been implicated in the pathophysiology of acute and chronic neurodegenerative conditions.
The invention further provides methods of treating or effecting prophylaxis of a disease mediated by excitotoxicity.
The exceptional significance of excitotoxicity to neuronal degeneration renders this area an important research target.
Volatile anesthetics may also protect against glutamate excitotoxicity and promote its uptake.
Taurine protects against excitotoxicity by blocking and reducing the overstimulation caused by excess glutamate.
The biochemical cascade resulting from ischemia and involving excitotoxicity is called the ischemic cascade.
Excitotoxicity is a complex process that involves significant changes in the cell cycle gene expression.
NMDA receptors are important for neuronal excitotoxicity and plasticity.
Cell excitotoxicity is a key event in the physiopathology of TBI.
Distinct roles of synaptic and extrasynaptic NMDA receptors in excitotoxicity.
Dose-dependent neuronal excitotoxicity of kainate.
CBD maintains calcium homeostasis and inhibits glutamate therefore decreasing excitotoxicity.
