Examples of 'ischemia-reperfusion' in a sentence
Meaning of "ischemia-reperfusion"
ischemia-reperfusion (noun): 'Ischemia-reperfusion' refers to the process of blood flow being temporarily restricted (ischemia) and then restored (reperfusion) in a particular organ or tissue. This term is commonly used in medical contexts, particularly in discussions about injuries, surgical procedures, and organ transplants
How to use "ischemia-reperfusion" in a sentence
Basic
Advanced
ischemia-reperfusion
Ischemia-reperfusion injury and hepatic encephalopathy.
Pathophysiology of ischemia-reperfusion and neuronal injury.
Ischemia-reperfusion injury is another indication.
This phenomenon is called ischemia-reperfusion injury.
Ischemia-reperfusion injury in human liver transplantation, mechanisms and effects on graft function.
Pathophysiology of ischemia-reperfusion myocardial injury.
Acute lung injury was induced by intestinal ischemia-reperfusion.
Such phenomenon is referred to as ischemia-reperfusion injury or simply as reperfusion injury.
In these experiments, antibodies were injection prior to induction of ischemia-reperfusion.
Histopathologic scoring of the ischemia-reperfusion injury to small intestine.
The situation resembles the problems encountered during ischemia-reperfusion.
In some embodiments, the ischemia-reperfusion event is related to myocardial infarction or stroke.
The role of mbl in ischemia and ischemia-reperfusion.
Alleviating ischemia-reperfusion injury improves the quality of life of patients undergoing reperfusion.
Another process that is intrinsically related to ischemia-reperfusion injury is apoptosis.
See also
Adenosine attenuates ischemia-reperfusion injury of the heart upon administration prior to ischemia or reperfusion.
There is also evidence that it attenuates murine ischemia-reperfusion injury.
Ischemia-reperfusion injury of the organs is also associated with vasculitis and autoimmune disease.
These cells are important in the pathophysiology of ischemia-reperfusion injury of organs.
The ischemia-reperfusion injury of the gut induces primary and secondary tissue damage.
It is believed that these deficits are due to oxidative stress caused by ischemia-reperfusion.
In an animal model, liver viability following ischemia-reperfusion was inversely related to cellular necrosis.
It has been proven that morphine has cardioprotective effects during ischemia-reperfusion.
The present invention can alleviate ischemia-reperfusion injury in any of such organs,.
The test for inhibiting the cardiac necrosis induced by a period of ischemia-reperfusion.
Preconditioning as a result of short episodes of ischemia-reperfusion leads to improved tissue resistance to ischemia.
Figure 7 is a multipanel figure showing nitrite therapy in myocardial ischemia-reperfusion injury.
Isoflurane induces protective effects during ischemia-reperfusion and lung injury induced by endotoxin or zymosan.
Figure 6 is a mutlipanel figure showing nitrite therapy in hepatic ischemia-reperfusion injury.
Ischemia-reperfusion injury could be prevented pharmacologically, and novel targets for this are needed.
The sham group was not subjected to the cold ischemia-reperfusion sequence.
The ischemia-reperfusion guides the activation and expression of adhesion molecules in endothelial cells and leucocytes.
The active antioxidant effect also reduces liver ischemia-reperfusion injury.
Ischemia-reperfusion was induced and 4 µg per gram of ALULA was injected intraperitoneally.
Tetrathiomolybdate for use in the treatment of ischemia-reperfusion injury of the heart.
The cardiac ischemia-reperfusion rat model also shows an increase in PDE1 activity.
Tetrathiomolybdate for use in the therapy of ischemia-reperfusion injury of the kidney.
There are no previous reports describing the development of hepatic steatosis after injury by ischemia-reperfusion.
During acute myocardial infarction the process of ischemia-reperfusion injury results in cell death.
Nitric oxide-donating compounds have shown promise as protective agents in experimental models of ischemia-reperfusion.
Understanding the physiological events involved in ischemia-reperfusion and leading to possible therapies is therefore critical.
One particular phenomenon which could be treated more effectively is ischemia-reperfusion injury.
Oxidative events following CNS ischemia-reperfusion are not limited to lipids.
This occurs both in syngeneic and allogeneic transplants and is a response to ischemia-reperfusion.
Additionally, they can be used for ischemia and ischemia-reperfusion injury resulting from shock and trauma.
One of the most intriguing applications for iNO is with lesions provoked by ischemia-reperfusion.
The role of TNF in ischemia-reperfusion injury of the brain was examined.
Therapeutic agent and therapeutic device for alleviating ischemia-reperfusion injury.
Liver ischemia-reperfusion I / R involves a complex series of processes that culminate in hepatocellular injury.
Cytoprotective effects of nitrite during in vivo ischemia-reperfusion of the heart and liver.
