Examples of 'lps-induced' in a sentence
Meaning of "lps-induced"
LPS-induced means induced or caused by lipopolysaccharides (LPS), which are large molecules found in the outer membrane of Gram-negative bacteria
How to use "lps-induced" in a sentence
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lps-induced
Inhibition of LPS-induced cytokine secretion in differentially treated DCs.
It seems to be a preferred and specific modulation of LPS-induced response.
A significant inhibition of the LPS-induced maturation of DCs was observed with secretome.
It seems to be a preferring and specifically modulation of LPS-induced response.
Percentage of inhibition of LPS-induced expression of costimulatory molecules in differentially treated DCs.
The composition for use of claim 1 whereby the pulmonary inflammation is LPS-induced.
Aminoguanidine treatment prevented the LPS-induced increases in plasma nitrate / nitrite levels.
The result indicates that the compounds of the present invention can restrain LPS-induced cytokine activity.
LPS-induced neutrophil accumulation in mice.
Also in lung tissue, plasma protein supplementation prevented LPS-induced leukocyte stimulation.
The lipid A analogs inhibited LPS-induced production of TNF in human whole blood in a concentration-dependent manner.
The compounds of this invention are active in vivo in the LPS-induced TNFa secretion model.
LPS-induced activation of DC is blocked by ManLAM.
Tipifarnib clearly showed global inhibition of LPS-induced inflammation in vitro and in vivo.
No LPS-induced changes were present in brain, heart, or small intestine.
See also
For example, the antibody binds to the target and neutralizes LPS-induced proinflammatory cytokine production.
Poir . extract on LPS-induced in vitro proliferation of splenocytes.
Subacute treatment 7 days with RESV was effective at preventing lipopolysaccharide LPS-induced lethality in mice.
Inhibition of LPS-induced TNF-release is taken as the readout.
Different batches of the composition were examined for their effect on LPS-induced release of TNF.
In condition of acute LPS-induced neuroinflammation, BH4 administration helps to reduce neuroinflammation.
Phosphatase inhibition by the disclosed PTP-B inhibitors reduces LPS-induced renal vascular leak.
LPS-induced activation of NF-kB and JNK appears to be independent of the presence of MyD88.
Effects of the peptides on LPS-induced TNF alpha production.
LPS-induced endotoxic shock.
FTIs may also be used to treat LPS-induced inflammation in particular.
LPS-induced acute inflammation.
Serum samples were tested for LPS-induced AP complement activation.
The results demonstrated that anti-CD200R antibody protects mice from LPS-induced toxicity.
In vivo testing for Example 1 LPS-induced neutrophilia in the mouse, duration of action.
Figure 5 shows in vivo hepcidin expression in the LPS-induced mouse model.
Fig. 6 illustrates reduction in LPS-induced cytotoxicity in a rASS dose-dependent fashion;.
The studies of the present invention additionally showed downregulation of LPS-induced MIP-1a by simvastatin.
Sialic Acid nanoparticles inhibit LPS-induced inflammatory response ( TNF alpha expression ).
LPS-induced secretion of TNFa was reduced by 40 % from these cells.
RESULTS Effect of simvastatin and tipifarnib on LPS-induced gene transcription.
IL-10 inhibits the LPS-induced expression of TNF-a RNA in a macrophage cell line.
Animal models demonstrate the inhibition of LPS-induced TNF-a production.
The kinetics of CGRP inhibition of LPS-induced sIg expression was examined in 70Z/3 cells.
Simvastatin and tipifarnib had contrasting effects on inhibition of LPS-induced IL-8 and IL-1B.
Attenuation of LPS-Induced Mortality.
Figures 5a-5e show the effect of simvastatin and tipifarnib on LPS-induced cytokine production in vivo.
For example, the antibodies are capable of blocking LPS-induced pro-inflammatory cytokine production.
Mice that do not express IRF3 are resistant to LPS-induced septic shock.
Figure 4 shows inhibition of LPS-induced IL-6 release by tipifarnib.
FIG . 5 shows in vivo hepcidin expression in the LPS-induced mouse model.
They significantly reduced IL-8 yield in the LPS-induced inflammatory response in the HT-29 cell line.
The results indicate that neutralization of IL-18 protects against LPS-induced myocardial dysfunction.
Human Whole Blood LPS-induced TNF-a assay.
Alkaline phosphatase inhibits the amplifying effect of ATP but not the LPS-induced IL-6 production itself.
