Examples of 'mice develop' in a sentence
Meaning of "mice develop"
mice develop: Describes the process of mice undergoing growth or change
How to use "mice develop" in a sentence
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mice develop
Aged apelin knockout mice develop progressive impairment of cardiac contractility.
Figure 7 illustrates with photomicrographs that adenosine deaminase partially-deficient mice develop liver fibrosis.
The mice develop the disease after being inoculated with a myelin protein.
The exocrine compartment is also altered as mice develop a pancreatitis phenotype.
These mice develop normally and do not display any gross abnormalities.
Researchers also observed that dominated mice develop atherosclerosis when they are still young.
The mice develop neurologic problems and die earlier than control mice.
Our preliminary data showed that these mice develop MS symptoms spontaneously.
ALS mice develop a motor disease that closely resembles ALS.
However, why only a subset of these mice develop leukemia was not understood.
Mice develop symptoms such as swollen wrists, ankles and digits.
However, why only a subset of these mice develop leukaemia was not understood.
The mice develop human-like tumor and liver metastases.
However, neither ace nor angiotensinogen null mice develop any overt heart disease.
The brains of these mice develop amyloid plaques and the mice are memoryimpaired.
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Three months after the immunization with the human ANCA, mice develop endogenous antibodies to ANCA.
First, all mice develop dense cortical cataracts.
Transgenic human Z-AAT expressing mice develop tumors with age.
These mice develop characteristics of metabolic syndrome, including insulin resistance, on a chow diet.
At late stage of tumour progression, MMTV-PyMT mice develop lung metastasis.
However desmin knockout mice develop normally and only experience defects later in life.
In addition, the results show that Annexin-1 deficient mice develop less severe EAE.
Despite this, the mice develop normal brains before dying immediately after birth.
This example also demonstrates that motor neurons of C9orf72 - / - mice develop significant mitochondrial dysfunction.
These mice develop plaques consisting of amyloid depositions in early age, starting at approximately 4 months.
The aromatase knock-out ( ARKO ) mice develop significant prostate hyperplasia.
These mice develop characteristics of AD including increases in beta-amyloid plaques and dystrophic neurites.
Indeed, in the absence of CD4 + T cells, mice develop a more pronounced and persistent inflammation.
Mice develop dengue viremia rising over several days, peaking on day 3 post-infection.
Over the subsequent 3-4 months, the mice develop renal glomerular fibrosis characteristic of human renal disease.
Germ-free mice develop more receptors for sweet flavors in their intestines, for example.
Cadherin-11 deficient mice develop less dermal fibrosis.
MRL / lpr mice develop massive splenomegaly and lymphadenopathy with disease progression.
With age, Tet2-deficient mice develop bona fide myeloid tumors.
Older mice develop hepatocellular carcinomas due to the sustained activation of peroxisome proliferator-activated receptor-alpha ( PPARα ).
Untreated Taiwan mice develop necrotic tails, which shorten over time.
H+T + mice develop clinical, biochemical, histological, and immunological features similar to human myositis.
Apo ( a ) transgenic mice develop lesions when fed a lipid-rich diet.
SOD1 mice develop ascending paralysis and exhibit early signs of the disease by 91 days.
Like humans, the mice develop problems with walking, lose muscle mass and eventually become paralyzed.
Most mice develop significant titers against Abeta42, the immunogen or against IAPP.
Interestingly, the mice develop T cell lymphomas independent of the Notch pathway.
Transgenic mice develop a deafness whose characteristics are similar to the ones of AUNA1.
These transgenic mice develop B-amyloid deposits at about 9 months of age.
MC4R null mice develop late onset obesity with hyperglycemia and hyperinsulinemia.
Example III MRL / lpr mice develop systemic lupus erythematosus ( SLE ) - like lesions.
OPG-deficient mice develop osteoporosis that can be rescued by injection of recombinant OPG.
Gondii-infected mice develop crohn ¿ s disease ( cd ) - like enteritis associated with severe systemic inflammatory response.
In general, MRL/1 mice develop major symptoms of membranoproliferative glomerulonephritis.
After TAC, mice develop heart failure within a period of 4 weeks.
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