Examples of 'myocardial oxygen' in a sentence
Meaning of "myocardial oxygen"
Myocardial oxygen: The amount of oxygen that is delivered to the heart muscle. It is essential for the heart to function properly and meet its energy demands
How to use "myocardial oxygen" in a sentence
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myocardial oxygen
Myocardial oxygen demand exceeds the supply.
These effects combine to reduce myocardial oxygen requirements.
They reduce myocardial oxygen demand and reverse coronary vasospasm.
It produce curative effect by reducing myocardial oxygen consumption.
Myocardial oxygen consumption.
The double product provides an estimate of myocardial oxygen consumption.
Myocardial oxygen consumption was shown as a crucial parameter in the response to rhythm troubles.
Exercise and mental stresses increase myocardial oxygen demand.
Myocardial oxygen consumption always increases as a result of anxiety T.
This leads to a reduction in cardiac workload and myocardial oxygen consumption.
Two important determinants of myocardial oxygen compensation are heart rate and pressure load.
Adenosine is an endogenous extracellular messenger that can regulate myocardial oxygen needs.
These actions combine to decrease myocardial oxygen demand and increase myocardial oxygen supply.
Both show a higher incidence of arrhythmias in patients with reduced myocardial oxygen supply.
Nitrates act primarily by reducing myocardial oxygen demand rather than increasing its oxygen supply.
See also
Such dilated arterioles may be unable to meet the increased myocardial oxygen demand.
Tachycardia as a predictor for increased myocardial oxygen consumption which doubles the incidence of myocardial ischemia.
The ultimate infarct size may be influenced by increases or decreases in myocardial oxygen utilization.
Persistent and severe interferences with myocardial oxygen supply can lead to acute myocardial infarction.
The key is to preserve effective tissue perfusion while avoiding excessive myocardial oxygen consumption.
SERV is an index of myocardial oxygen supply and demand calculated through PWA.
Stable angina pectoris is caused by the increased myocardial oxygen demand in most cases.
Myocardial oxygen consumption. while avoiding excessive.
The key is to preserve effective tissue perfusion while avoiding excess myocardial oxygen consumption.
Despite this reduction, myocardial oxygen consumption did not change.
Blood pressure and increase coronary blood flow and increase myocardial oxygen consumption.
During an ischemic event, myocardial oxygen demand exceeds the supply.
Levosimendan is a novel inotropic agent that improves cardiac contractility without increasing myocardial oxygen consumption.
It also raises myocardial oxygen demands as torsional defamation ( strain ) increases.
All these conditions entail a mismatch between coronary blood inflow and myocardial oxygen demand.
Further, amlodipine has been shown to increase myocardial oxygen supply by dilating the coronary arteries.
By decreasing heart rate, blood pressure and myocardial contractility, betablockers reduce myocardial oxygen uptake.
Furthermore, reduced myocardial oxygen supply may cause ischemia, and worsened cardiovascular ventricular dysfunction.
Cardiac output Increased output and myocardial oxygen demand.
Increased output and myocardial oxygen demand Increased with acute dose, decreased with chronic use.
Subsequently, left ventricular work and myocardial oxygen demand decrease.
Myocardial oxygen consumption ( MO2 ) depends on the double product.
As a result, ischemia may occur at a lower level of myocardial oxygen demand.
Myocardial oxygen consumption . FIG.
For example, during exercise or with mental stress, myocardial oxygen demands increase.
Myocardial oxygen consumption ( MVO2 ) was calculated using the formula of Rooke and Feigl.
Levosimendan improves coronary blood flow, decreases myocardial oxygen extraction, and improves ischemic myocardium performance.
Through its inotropic and vasodilator effect, levosimendan enhances cardiac output without increasing myocardial oxygen requirements.
Specifically, these agents help to decrease myocardial oxygen consumption ( which is increased by adrenergic agents ).
These B-blockers prevent angina primilarly by lowering myocardial oxygen demand.
Furthermore, myocardial oxygen consumption was decreased in a dose-dependent manner ( FIG . 5 ).
Trimetazidine exerts its anti-ischemic properties without affecting myocardial oxygen consumption and blood supply.
Furthermore, myocardial oxygen consumption was decreased in a dose-dependent manner ( Figure 5 ).
In vitro, ACE inhibitors cause an increase in myocardial oxygen utilisation.
It reduces the preload, the left ventricular end-diastolic volume, and, consequently, the myocardial oxygen consumption.
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