Examples of 'neuronal damage' in a sentence

Meaning of "neuronal damage"

neuronal damage: This term describes harm or injury to the neurons in the brain or nervous system, which can result in various cognitive or physical impairments

How to use "neuronal damage" in a sentence

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neuronal damage
Deposition of these plaques results in neuronal damage.
Ischemic neuronal damage.
This protein is commonly used as a biomarker of neuronal damage.
As well as neuronal damage.
Research is also carried out regarding early markers to prevent neuronal damage.
Neuronal damage was assessed as described previously Pringle et al.
May be useful for preventing ischemic neuronal damage.
Neuronal damage was assessed using ImageJ running on a PC.
There are three possible methods in which neuronal damage occurs.
Holo-transferrin increases neuronal damage in in vitro and in vivo brain stroke models.
Applicants preferred utility is the prevention of ischemic or neuronal damage following a stroke.
The severity of the neuronal damage is reflected in the amplitude of the GABA-mediated depolarization.
It is caused by neuronal damage.
The focus has been on controlling calcium dysfunction of free radical mediated neuronal damage.
Vitamin K is essential to stop neuronal damage in the brain.

See also

Other studies have linked elevated levels of this hormone to increased neuronal damage.
However, strokes frequently cause permanent neuronal damage or result in the death of the individual.
Moreover, choline is neuroprotective in a variety of experimental models of neuronal damage.
Furthermore, the cartilage degradation leads to chronic neuronal damage with neuropathic-like characteristics.
Activity of the compounds of formula 1 is illustrated by a decrease in neuronal damage.
This continuous mild activation may lead to neuronal damage and impairment of synaptic plasticity ( learning ).
Excessive production of these inflammatory mediators could cause neuronal damage and death.
Gamma-lon is a remedy for preventing neuronal damage to the brain and restoring the neural connections;.
Role pf TRP channels in anoxic neuronal damage.
Xenon attenuated neuronal damage induced by N-methylD-aspartate NMDA administration in rats.
For example, such inhibitors can reduce neuronal damage caused by epilepsy.
Initially, degeneration of the injured nerve is probably attributable to direct neuronal damage.
It also limits neuronal damage in the brain in patients with Alzheimer 's disease.
In other words, it is known that it may cause so-called ischemic neuronal damage.
Neuronal damage was assessed by densitometric quantification of PI uptake in area CA1 and CA3.
Hence, sudden interruption of glucose delivery to the brain results in neuronal damage.
Omega-3 and the reduction in neuronal damage secondary to traumatic brain injury.
The amalgamation of solid phase / combinatorial chemistry and in vitro models of neuronal damage e . g.
In vivo neuronal damage was validated using magnetic resonance imaging ( MRI ).
In a preferred embodiment, the neuroprotection is against neuronal damage resulting from an impact trauma.
Furthermore, these increased levels of quinolinic acid are great enough to produce excitotoxic neuronal damage.
Use according to claim 5, wherein the neuronal damage results from Parkinson 's disease.
The sustained-release preparation of the invention is useful to prevent or treat neuronal damage.
Neuronal damage was assessed by densitometric quantification of propidium-iodide ( PI ) uptake Fig.
Even if transient, both global and focal ischemia can produce widespread neuronal damage.
Untreated rats routinely demonstrate ischemic neuronal damage after 20 or 30 minutes of 4-vessel occlusion.
Even if transient, both global and focal ischemia can cause widespread neuronal damage.
More specifically, they help counteract glutamate excitoxicity, which causes neuronal damage following a brain injury.
This leads to priming / activation of microglial cells and the adverse repercussions leading to neuronal damage.
It may help Alzheimer 's disease patients by limiting neuronal damage in the brain.
We have discovered that certain compounds protect neurons against NMDA receptor-mediated neuronal damage.
Thus, NSE may serve as a quantitative marker for the degree of neuronal damage.
Figure 2 illustrates the effect of IL-1 receptor antagonist protein on NMDA receptor mediated neuronal damage.
Intriguingly, even a few hours of mild hyperoxia ( 30 % ) were sufficient to cause neuronal damage.

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