Examples of 'neurotransmitter release' in a sentence

Meaning of "neurotransmitter release"

Neurotransmitter release refers to the process by which neurotransmitters, chemical substances that transmit signals between nerve cells in the brain and nervous system, are released from one neuron to another. This release allows for the communication and transmission of electrical signals within the nervous system, playing a crucial role in various physiological and cognitive functions, including mood regulation, motor control, and sensory perception

How to use "neurotransmitter release" in a sentence

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neurotransmitter release
Pharmacological studies of opioid action on neurotransmitter release.
An increase or decrease in neurotransmitter release also could be measured to monitor the response.
The neurotoxins are still able to inhibit a neurotransmitter release.
NCCs regulate neurotransmitter release in specific neuronal populations responsible for the spinal processing of pain.
It is also used in scientific studies of neurotransmitter release in neurons.
Enhanced neurotransmitter release should reverse the symptoms associated with depression and Alzheimers disease.
Note that the change in neurotransmitter release is by rods.
Elevation of intracellular calcium concentration is required for neurotransmitter release.
Another cause of a disturbed neurotransmitter release is represented by drug abuse.
Activation of nAChRs by nicotinic compounds results in neurotransmitter release.
The normal electrical response to neurotransmitter release is a measure of synaptic strength.
This provides a role for palmitoylation in regulating neurotransmitter release.
Spatial and temporal plasticity of neurotransmitter release at a developing central synapse.
Certain of those effects can be related to effects upon neurotransmitter release.
Neurotransmitter release assay Methods.

See also

Said neurotoxins are still able to inhibit neurotransmitter release.
An increase in neurotransmitter release follows traumas such as hypoxia, ischemia and seizures.
Calcium channels play a central role in neurotransmitter release.
An increase in neurotransmitter release follows traumas such as hypoxia, ischaemia and seizures.
Molecular mechanisms of neurotransmitter release.
Neurotransmitter release is calculated as a fraction of total radioactivity present in the well . Results.
Both toxins function by inhibiting neurotransmitter release in affected neurons.
Isolated tissue assays are traditional pharmacological methods for measuring effects on neurotransmitter release.
Presynaptic neurotoxins disrupt neurotransmitter release from the axon terminal.
This reduces the frequency of action potentials which reduces neurotransmitter release.
Active omega-conopeptides also inhibit neurotransmitter release in various regions of the nervous system.
This may lead to a disinhibtory action and enhanced neurotransmitter release.
These toxins act by blocking neurotransmitter release from neurons, thus paralyzing animals and humans.
These ion channel interactions result in lowered neurotransmitter release.
FIG . 1A shows a schematic for the neurotransmitter release mechanism of a central and peripheral neuron.
Drug abuse represents another cause of impaired neurotransmitter release.
Function, blocking neurotransmitter release by hydrolysis of snap25.
Certain of those effects may be related to effects upon neurotransmitter release.
Figures 4-6 show inhibition of neurotransmitter release from cultured neuronal cells.
Receptors work together and lead to depolarization of the cell and neurotransmitter release.
Botulinum neurotoxins can block neurotransmitter release for several months.
The resulting accumulation of positive charges depolarizes the cell and leads to neurotransmitter release.
Other sources of weakness may be secondary to neurotransmitter release dysfunction at the synaptic terminal.
The neurotoxin may alleviate the symptom that is associated with hyperactive neurotransmitter release.
Inhibition of neurotransmitter release by several neuroprotective anesthetic agents ( isoflurane, etomidate, propofol ) was also known.
Nicotine has been reported to have en effect on neurotransmitter release in several systems.
Typically, the neurotransmitter release inhibition effect can be expected to wear off within about 2-4 months.
The depolarization of the hair cells in both instances causes signal transduction via neurotransmitter release.
The a2C-adrenoceptor is particularly suited to control neurotransmitter release at low action potential frequencies.
MGluR5 receptors can therefore modify the post-synaptic responses to neurotransmitter or regulate neurotransmitter release.
In addition, calcium also serves to facilitate neurotransmitter release and muscle contraction.
GABAB receptor-mediated Ca2 + channel inhibition is the principle mechanism for presynaptic inhibition of neurotransmitter release.
Figure 2, Alteration of neurotransmitter release.
Galanin is predominantly an inhibitory, hyperpolarizing neuropeptide [ 1 ] and as such inhibits neurotransmitter release.
Fig . s 4-6 show inhibition of neurotransmitter release from cultured neuronal cells ; and.

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