Examples of 'proinflammatory' in a sentence
Meaning of "proinflammatory"
proinflammatory (adjective) - describing a substance, condition, or response that promotes inflammation in the body. It is often used in medical, scientific, or health-related contexts to refer to exacerbating factors
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- Producing or promoting inflammation.
- An inflammatory substance.
How to use "proinflammatory" in a sentence
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proinflammatory
A proinflammatory cytokine may be present in adipose tissues.
Cellular activation leads to release of proinflammatory mediators.
Nitric oxide is a proinflammatory mediator produced by macrophages.
It also demonstrates pyrogenic and proinflammatory effects.
Promotes production of proinflammatory cytokines by monocytes and macrophages.
Leptin receptor is also upregulated by proinflammatory signals.
Proinflammatory components of certain bacterial strains have been identified.
Each haplotype results in a net proinflammatory response.
Proinflammatory cytokines are probably involved in genesis and perpetuation of disease.
Stress can activate proinflammatory pathways.
Proinflammatory cytokines have also been implicated as mediators of transplant rejection.
This in turn produces a proinflammatory and hypercoagulable state.
Neuroinflammation involves activation of microglia and production of proinflammatory cytokines.
It is characterised by an early proinflammatory response and followed by a state of immunosuppression.
It is a chemical which inhibits a wide range of proinflammatory proteins.
See also
Analysis of proinflammatory cytokines to evaluate the immune response of peptides.
Leptin acts on the immune system as a proinflammatory cytokine.
T helper cells produce proinflammatory cytokines to help the defending cells.
Pathogenesis is due in part to the secretion of proinflammatory cytokines.
Desflurane may increase proinflammatory cytokine expression in the alveolar macrophages.
According to other sources escin has proinflammatory effects.
The relationship between proinflammatory mediator expression and diaphragm dysfunction.
It acts by decreasing oxidative stress and proinflammatory biomolecules.
Other proinflammatory cytokines have been correlated with a variety of disease states.
Tildrakizumab inhibits the release of proinflammatory cytokines and chemokines.
These proinflammatory cytokines play essential roles in pathogenesis of rheumatoid arthritis.
These substances are proinflammatory mediators.
Proinflammatory cytokines and prostanoids have been implicated in the development of myositis.
Any mammalian cell that produces proinflammatory cytokines is intended.
Some research indicates that animal protein may be somewhat proinflammatory.
It inhibits the release of proinflammatory cytokines from activated immune cells.
Both of them have a role in the expression of proinflammatory cytokines.
The underlying proinflammatory response affects all the organs simultaneously.
These cells release histamine and other proinflammatory mediators.
Proinflammatory cytokines are candidates as the initial causative agents of the metabolic changes in tb.
The oxygen radicals are almost all proinflammatory and profibrogenic.
Proinflammatory cytokines are among the most powerful stimulants of bone resorption known.
Part of the neuroprotective effect of hypothermia is due to proinflammatory pathway blockade.
Excessive or persistent proinflammatory cytokine production plays a central role in autoimmune diseases.
These changes may be associated with a prothrombotic and proinflammatory state.
The chronic overproduction of proinflammatory cytokines leads to the generation of free radicals.
This kinase is activated by various environmental stresses and proinflammatory cytokines.
This shows that the prothrombotic and proinflammatory states may be metabolically interconnected.
Eosinophil depletion does not produce chronic systemic alterations of proinflammatory cytokines.
It was suggested that this proinflammatory cytokine actively contributes to synovial inflammation in rheumatoid arthritis.
Free heme and hemin are cytotoxic and proinflammatory compounds.
This proinflammatory process is the probable causal agent of the reversible obstruction of lower airways.
Lipopolvsaccharide was used to stimulate and increase the secretion of proinflammatory cytokines.
Desflurane has been noted to produce more proinflammatory response compared with the sevoflurane.
Lesions were preceded by the expression and progressive upregulation of proinflammatory molecules.
