Examples of 'tumorigenesis' in a sentence
Meaning of "tumorigenesis"
Tumorigenesis is the process by which tumors are formed. It refers to the development of cancerous tumors
Show more definitions
- Production of a new tumor or tumors.
- The process involved in the production of a new tumor or tumors.
How to use "tumorigenesis" in a sentence
Basic
Advanced
tumorigenesis
Met functions in tumorigenesis and tumor metastasis.
This protein may play a role in tumorigenesis.
Menin and endocrine tumorigenesis and osteoblast function.
The protein may play a role in tumorigenesis.
Tumorigenesis appears to coincide with the onset of angiogenesis.
Some of the candidates have been implicated in tumorigenesis.
Mechanisms of tumorigenesis and tumor progression research group.
Another gene known to be involved in tumorigenesis is KRas.
Tumorigenesis or a graft versus host reaction.
Aberrant activation of this pathway contributes to tumorigenesis.
Tumorigenesis is a largely continuous occurrence in animals such as humans.
This mechanism might contribute to tumorigenesis.
The mechanism of tumorigenesis in these cancer syndromes is still contentious.
Its serum levels are associated with colon tumorigenesis.
Tumorigenesis develops aggressively and the therapeutic alternatives remain limited.
See also
Tipping the scales to initiate tumorigenesis in the colon.
Genomic deletions have long been considered to be an important factor in tumorigenesis.
Osteonectin inhibits tumorigenesis in mice.
Losses of these major control elements are hallmarks of tumorigenesis.
Senescence limits tumorigenesis and tissue damage throughout the lifetime.
The formation of tumors is called tumorigenesis.
Tumor formation or tumorigenesis is the initial sign of the appearance of cancer.
The methods are also useful to prevent tumorigenesis in a subject.
During tumorigenesis cancer cells acquire the ability to evade this immunosurveillance by multiple mechanisms.
Another gene known to be involved in tumorigenesis is mTOR kinase.
Identification of genes predisposing to tumor syndromes has raised general awareness of tumorigenesis.
Autophagy seems to have a dichotomous role in tumorigenesis and tumor progression.
The efficacy of compounds disclosed herein was evaluated in a standard murine model of tumorigenesis.
This feature play significant role in tumorigenesis because angiogenesis usually precede metastasis.
This uncontrolled growth is a step in tumorigenesis.
The following example shows that tumorigenesis alters CystC expression in human tissues.
Gingerols may interfere with tumorigenesis.
Tumorigenesis assays were performed as described in Nam et al.
Inflammatory cell infiltrates are associated with different stages of tumorigenesis.
The role of RA synthesis in mammary tumorigenesis has been poorly characterized.
Notably the mutant mice also showed increases in tumorigenesis.
LPA contributes to tumorigenesis by increasing motility and invasiveness of cells.
Treatments can be targeted to aberrant molecules driving the tumorigenesis.
There was no evidence of tumorigenesis associated with Modafinil administration in these studies.
Both pathways are implicated in tumorigenesis.
Exemplary genes involved in tumorigenesis include the genes encoding DNA methyltransferases.
These changes in transcription are thought to be the basis for tumorigenesis.
A general theme in carcinogenesis and tumorigenesis is the down regulation of the GJIC.
Exposure to aristolochic acid is associated with a high incidence of uroepithelial tumorigenesis.
Activated Notch signaling is known to induce tumorigenesis in various tumor models.
Its already known that increased expression of these markers is related to tumorigenesis.
RTKs overactivation induces cell transformation and tumorigenesis notably by promoting survival.
Choline kinase alpha therefore is a good potential therapeutic target in human tumorigenesis.
Two biological processes commonly involved in tumorigenesis include gene amplification and DNA methylation.
Accumulating evidence indicates that miRNAs have a causal role in tumorigenesis.
