Examples of 'beta-catenin' in a sentence
Meaning of "beta-catenin"
Beta-catenin (noun) - A protein that plays a crucial role in cell adhesion and signaling pathways. It is involved in various cellular processes, including embryonic development and tissue regeneration
How to use "beta-catenin" in a sentence
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beta-catenin
Beta-catenin is widely expressed in many tissues.
Modulating hedgehog and beta-catenin signaling in osteoarthritis.
Beta-catenin serves as the loading control.
This inhibits the expression of cadherins and beta-catenin.
Beta-catenin expression appeared on the cell membranes.
Disclosed is that the amount or activity of beta-catenin in an individual is modulated.
Beta-catenin mutations are commonly found in endometrial cancers with squamous cells.
Disclosed is that the amount or activity of beta-catenin in a sample is measured.
Beta-catenin is a cytoskeletal component that enters the nucleus to act as a transcriptional cofactor.
Altered expression profiles in beta-catenin have been associated with dilated cardiomyopathy in humans.
Such antimicrobial gene expression was synergized following co-stimulation by calyculin that promotes beta-catenin degradation.
These results show that beta-catenin play a role in the adriamycin induced podocyte injury.
Control of kidney cell determination and morphogenesis by BMPs and Beta-catenin.
Active beta-catenin signaling stood out as the primary difference between the two groups.
The agent may act by mimicking the activity of beta-catenin in the individual 's heart.
See also
It also is a direct target gene of the Wnt pathway and is upregulated by beta-catenin.
These results show that that beta-catenin functions differently at different stages of fracture repair.
Methods and compositions for the specific inhibition of beta-catenin by double-stranded RNA.
These mutations generally affect beta-catenin phosphorylation and stability, hindering its degradation vía the ubiquitin pathway.
Injection of NIK suppressed the induction of axis duplication by microinjected beta-catenin in Xenopus embryos.
Wnt stabilizes beta-catenin which then binds to TCF and activates gene transcription.
Researchers then wanted to see how suppression of the beta-catenin protein impaired human leukemic cells.
Phosphorylation of Beta-catenin induces the ubiquitination and proteolytic degradation of beta-catenin by the proteasome.
Researchers then wanted to see how suppression of the beta-catenin protein impaired human leukaemic cells.
Non-phosphorylated beta-catenin is stable and enters the nucleus to regulate transcription with TCF.
The ARM domains play a role in mediating the interaction of beta-catenin with its ligand.
And induced by Wnt or beta-catenin signaling Chesire et al.
This is entirely consistent with our epistasis analysis that places Trabid 's function below activated beta-catenin.
Beta-catenin alters T cells in lasting and harmful ways.
In adenomas, the staining of beta-catenin was significantly p.
Beta-catenin in aggressive fibromatosis.
In one embodiment, the activator is beta-catenin.
Inhibition of beta-catenin expression in cancer cells ; and.
In certain embodiments, the agents disrupt beta-catenin signaling.
Numerous studies have linked beta-catenin pathway activation to tumor initiation, maintenance, and growth.
In another embodiment, the activator is a beta-catenin related protein.
Beta-catenin is a proto-oncogene.
When wnt ligand is absent, beta-catenin is being destroyed.
Sections are also probed with antibodies to c-myc and beta-catenin.
Actin binds a-catenin which binds beta-catenin which in turn binds E-cadherin.
MUC1 cytoplasmic tail was shown to interact with Beta-catenin.
RSPO1-Fc induced luciferase activity from the beta-catenin responsive promoter in a dose dependent manner.
The first two catenins that were identified [ 2 ] became known as alpha-catenin and beta-catenin.
Wnt3a induces the accumulation of nuclear beta-catenin in lung mesenchymal cells.
Beta-catenin ( B-catenin ) is part of a complex of proteins that constitute adherens junctions ( AJs ).
This interaction is antagonized by degradation of beta-catenin by GSK3B.
However, re-expression of TCF4 and beta-catenin does not overcome the Trabid requirement of these cells.
It is shown that this treatment profoundly inhibits the constitutive beta-catenin / Tcf4 activity.
Example 8 . rSIFN-co decreased beta-catenin protein level in cancer cells.
But CCL4 expression is suppressed by tumor cells with high beta-catenin levels . ”.
