Examples of 'tnf-alpha' in a sentence
Meaning of "tnf-alpha"
tnf-alpha (noun): Tumor necrosis factor-alpha, a cell signaling protein involved in inflammation, immune response, and cell death. TNF-alpha plays a crucial role in regulating the body's response to infection, injury, and diseases such as rheumatoid arthritis, Crohn's disease, and psoriasis
How to use "tnf-alpha" in a sentence
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tnf-alpha
However TNF-alpha has deleterious effects.
Inhibition of the cytotoxic activity of TNF-alpha.
Polymorphism of TNF-alpha in autoimmunity and tuberculosis.
Recombinant antibodies specific for TNF-alpha.
Inhibition of binding of TNF-alpha to its receptor.
Infliximab works by blocking the action of TNF-alpha.
Concurrent administration of TNF-alpha inhibitor and abatacept.
Hairy cells produce and thrive on TNF-alpha.
Concurrent administration of TNF-alpha inhibitor and anakinra.
Such inflammatory factors include interleukins and TNF-alpha.
Inhibition of the attachment of TNF-alpha to its membrane receptor.
The oxidized TNF-alpha is further purified by ion exchange chromatography.
Substituted phenethylsulfones and method of reducing tnf-alpha levels.
Other signals include TNF-alpha and lipopolysaccharides from bacteria.
In the immune cells it mediates parts of the responses to TNF-alpha.
See also
An inflammatory compound called TNF-alpha appears to play a major role.
A TNF-alpha converting enzyme has been shown to mediate this conversion.
They are typically used when TNF-alpha drugs have been ineffective.
People with certain conditions have too much TNF-alpha.
Results are expressed as % inhibition of mean TNF-alpha concentration in control mice.
Etanercept has the potential to treat these diseases by inhibiting TNF-alpha.
It is well known that prolonged TNF-alpha treatment by itself is highly toxic.
Includes study in patients who were inadequate responders to or intolerant of TNF-alpha inhibitors.
Excessive or unregulated TNF-alpha production has been implicated in a number of disease conditions.
There are numerous examples of disorders in which TNF-alpha activity is detrimental.
Inhibition of TNF-alpha - a therapeutic alternative for chronic treatment resistant ocular inflammation.
Formulation of human antibodies for treating TNF-alpha associated disorders.
Human TNF-alpha is active on murine cells with a slightly reduced specific activity.
In contrast to its effect on cell migration, TNF-alpha downregulate cell proliferation.
The line depicts the positive control value of cynomolgus TNF-alpha.
In addition, we observed increase in TNF-alpha levels in both groups after the treatment.
Compounds of the invention are inhibitors of tumor necrosis factor, especially TNF-alpha.
The causal relation between TNF-alpha blockade and neutropenia is still poorly understood.
In this study, there is no genetic abnormality in the TNF-alpha regulatory pathway.
Values for TNF-alpha are calculated from a recombinant murine TNF-alpha standard curve.
There are several nutrients that decrease TNF-alpha levels in the body.
In a further embodiment, a kit according to the can comprise a polynucleotide encoding TNF-alpha.
The signaling pathways by which tnf-alpha acts upon the parasite remain unknown.
Kits useful according to the invention can include an isolated TNF-alpha.
Luteolin also inhibits excess of TNF-alpha production, the direct cause of the inflammation.
A known concentration of cells is added to each well followed by addition of TNF-alpha.
The inhibition of TNF-alpha further inhibits the migration and maturation of dendritic cells.
The line depicts the positive control value of cynomologous TNF-alpha.
The presence of TNF-alpha inhibits the cells from growing through induction of apoptosis.
The mechanism of inhibition occurs at a post-translational step in TNF-alpha production.
Immune cells produce TNF-alpha when recognising salmonella, thus alarming other immune cells.
There were no statistically significant differences between the TNF-alpha levels of these subsets.
Values for TNF-alpha were calculated from a recombinant murine TNF-alpha standard curve as above.
This was concomitant with lower levels of pro-inflammatory mediators such as TNF-alpha.
There is evidence that overproduction of TNF-alpha may play a major pathogenic role in several diseases.
